切换至 "中华医学电子期刊资源库"

中华产科急救电子杂志 ›› 2012, Vol. 01 ›› Issue (02) : 140 -143. doi: 10.3877/cma.j.issn.2095-3259.2012.02.016

所属专题: 文献

综述

正常妊娠和子痫前期对肾脏的影响研究进展
韩磊1, 张欣1, 李力1,()   
  1. 1. 400042 重庆第三军医大学大坪医院野战外科研究所妇产科
  • 收稿日期:2012-09-30 出版日期:2012-11-18
  • 通信作者: 李力
  • 基金资助:
    国家自然科学基金(81170576)

Update of effect on the kidney in normal pregnancy and preeclampsia

Lei HAN1, Xin ZHANG1, Li LI1()   

  • Received:2012-09-30 Published:2012-11-18
  • Corresponding author: Li LI
引用本文:

韩磊, 张欣, 李力. 正常妊娠和子痫前期对肾脏的影响研究进展[J/OL]. 中华产科急救电子杂志, 2012, 01(02): 140-143.

Lei HAN, Xin ZHANG, Li LI. Update of effect on the kidney in normal pregnancy and preeclampsia[J/OL]. Chinese Journal of Obstetric Emergency(Electronic Edition), 2012, 01(02): 140-143.

[1]
Cornelis T, Odutayo A, Keunen J, et al. The kidney in normal pregnancy and preeclampsia. Semin Nephrol, 2011, 31(1):4-14.
[2]
Jeyabalan A, Novak J, Danielson LA, et al. Essential role for vascular gelatinase activity in relaxin-induced renal vasodilation, hyperfiltration, and reduced myogenic reactivity of small arteries. Circ Res, 2003,93(12):1249-1257.
[3]
Hladunewich MA, Derby GC, Lafayette RA, et al. Effect of L-arginine therapy on the glomerular injury of preeclampsia: a randomized controlled trial. Obstet Gynecol, 2006,107(4):886-895.
[4]
Zhou CC, Ahmad S, Mi T, et al. Autoantibody from women with preeclampsia induces soluble Fms-like tyrosine kinase-1 production via angiotensin type 1 receptor and calcineurin/nuclear factor of activated T-cells signaling. Hypertension, 2008, 51(4):1010-1019.
[5]
Anton L, Merrill DC, Neves LA, et al. The uterine placental bed Renin-Angiotensin system in normal and preeclamptic pregnancy. Endocrinology, 2009, 150(9):4316-4325.
[6]
Jeyabalan A, Conrad KP. Renal function during normal pregnancy and preeclampsia. Front Biosci, 2007,12:2425-2437.
[7]
Gilbert JS, Babcock SA, Granger JP. Hypertension produced by reduced uterine perfusion in pregnant rats is associated with increased soluble fms-like tyrosine kinase-1 expression. Hypertension, 2007, 50(6):1142-1147.
[8]
Garovic VD. The role of angiogenic factors in the prediction and diagnosis of preeclampsia superimposed on chronic hypertension. Hypertension, 2012, 59(3):555-557.
[9]
Crispi F, Llurba E, Dominguez C, et al. Predictive value of angiogenic factors and uterine artery Doppler for early- versus late-onset pre-eclampsia and intrauterine growth restriction. Ultrasound Obstet Gynecol, 2008, 31(3):303-309.
[10]
Verlohren S, Stepan H, Dechend R. Angiogenic growth factors in the diagnosis and prediction of pre-eclampsia. Clin Sci (Lond), 2012,122(2):43-52.
[11]
Espinoza J, Romero R, Nien JK, et al. Identification of patients at risk for early onset and/or severe preeclampsia with the use of uterine artery Doppler velocimetry and placental growth factor. Am J Obstet Gynecol, 2007,196(4):326 e321-e313.
[12]
Stillman IE, Karumanchi SA. The glomerular injury of preeclampsia. J Am Soc Nephrol, 2007,18(8):2281-2284.
[13]
Maynard SE, Thadhani R. Pregnancy and the kidney. J Am Soc Nephrol, 2009, 20(1):14-22.
[14]
Mirza FG, Cleary KL. Pre-eclampsia and the kidney. Semin Perinatol, 2009, 33(3):173-178.
[15]
Fischer MJ. Chronic kidney disease and pregnancy: maternal and fetal outcomes. Adv Chronic Kidney Dis, 2007,14(2):132-145.
[16]
Benton SJ, Hu Y, Xie F, et al. Angiogenic factors as diagnostic tests for preeclampsia: a performance comparison between two commercial immunoassays. Am J Obstet Gynecol, 2011, 205(5):469 e461-e468.
[17]
Naresh CN, Hayen A, Craig JC, et al. Day-to-day variability in spot urine protein-creatinine ratio measurements. Am J Kidney Dis, 2012, 60(4):561-566.
[18]
Brown MA. Pre-eclampsia: Proteinuria in pre-eclampsia-does it matter any more?. Nat Rev Nephrol, 2012, 8(10):563-565.
[19]
Foster RR, Saleem MA, Mathieson PW, et al. Vascular endothelial growth factor and nephrin interact and reduce apoptosis in human podocytes. Am J Physiol Renal Physiol, 2005, 288(1):F48-F57.
[20]
Lindheimer MD, Kanter D. Interpreting abnormal proteinuria in pregnancy: the need for a more pathophysiological approach. Obstet Gynecol, 2010,115(2 Pt 1):365-375.
[21]
Imbasciati E, Gregorini G, Cabiddu G, et al. Pregnancy in CKD stages 3 to 5: fetal and maternal outcomes. Am J Kidney Dis, 2007, 49(6):753-762.
[22]
Collino F, Bussolati B, Gerbaudo E, et al. Preeclamptic sera induce nephrin shedding from podocytes through endothelin-1 release by endothelial glomerular cells. Am J Physiol Renal Physiol, 2008, 294(5):F1185-F1194.
[23]
Garovic VD, Wagner SJ, Turner ST, et al. Urinary podocyte excretion as a marker for preeclampsia. Am J Obstet Gynecol, 2007, 196(4):320 e321-e327.
[24]
Piccoli GB, Attini R, Vasario E, et al. Pregnancy and chronic kidney disease: a challenge in all CKD stages. Clin J Am Soc Nephrol, 2010,5(5):844-855.
[25]
Phelan ST. Renal disease in pregnancy ambulatory issues. Clin Obstet Gynecol, 2012, 55(3):829-837.
[26]
Rashid M, Rashid HM. Chronic renal insufficiency in pregnancy. Saudi Med J, 2003, 24(7):709-714.
[27]
van Pampus MG, Aarnoudse JG. Long-term outcomes after preeclampsia. Clin Obstet Gynecol, 2005, 48(2):489-494.
[28]
Ruiz-Irastorza G, Khamashta MA. Lupus and pregnancy: ten questions and some answers. Lupus, 2008,17(5):416-420.
[29]
Egerman RS, Ramsey RD, Kao LW, et al. Hypertensive disease in pregnancies complicated by systemic lupus erythematosus. Am J Obstet Gynecol, 2005,193(5):1676-1679.
[30]
Seeman T. Hypertension after renal transplantation. Pediatr Nephrol, 2009, 24(5):959-972.
[31]
Perales-Puchalt A, Vila Vives JM, Lopez Montes J, et al. Pregnancy outcomes after kidney transplantation-immunosuppressive therapy comparison. J Matern Fetal Neonatal Med, 2012, 25(8):1363-1366.
[32]
Cooper WO, Hernandez-Diaz S, Arbogast PG, et al. Major congenital malformations after first-trimester exposure to ACE inhibitors. N Engl J Med, 2006, 354(23):2443-2451.
[33]
Hladunewich MA, Myers BD, Derby GC, et al. Course of preeclamptic glomerular injury after delivery. Am J Physiol Renal Physiol, 2008, 294(3):F614-F620.
No related articles found!
阅读次数
全文


摘要


AI


AI小编
你好!我是《中华医学电子期刊资源库》AI小编,有什么可以帮您的吗?